How Anabolic Steroids Affect Your Sex_Drive and the role of estrogen, DHT, and progesterone in determining their affects.
Roid Use or No Roid Use: Guard Your Testosterone Levels and Control Estrogen for Optimal Sex-Drive
Murder of Sex_Drive
Your libido is, well, essential. That is an understatement. If you can’t feel like a man, no use looking like the best built one. Okay, some things are known about male sex_drive, many things are not. We will focus on what we do know, and, what the majority of cases I’ve seen have taught me. We have already mentioned much of the effect of DHT on the sex_drive in the DHT section, however, we can expand a bit and give you some more useful and practical information. With respect to anabolic steroids, the male sex_drive is affected by the following.
I. The androgenicity of the drug(s) used.
II. The aromatizing qualities of the drug(s) used.
III. The progestagenic influence of the drug.
IV. The total amount of drug(s) in the system at any give time.
I. First, the androgenicity of the drug. In general, anabolic steroids that are highly androgenic have an intense affect on the libido. Authors will argue expertise about which drugs are more and less androgenic. You’ll read that deca is moderate, then you’ll read its very androgenic, you’ll read that winstrol is anabolic, then you’ll read its more androgenic…bla, bla, bla.
Here is the empirical facts in most (again not all, and there are some exceptions) cases. Testosterone makes you horny. Unless you have been on a cycle forever with no break, the first few days and weeks of test use definitely increase sex_drive. Dianabol is also a popular one for making juicers super horny. These drugs in almost all of the literature are known “androgenic drugs”. These are two no brainers. This is just the ground level illustration of anabolic steroids having a positive affect on sex_drive.
Nandrolone, as we’ve mentioned has a lessening affect on the sex_drive. This is variable from person to person as we’ve stated previously, but is largely related to dosage. The reasons why we have discussed in the DHT section. Whether or not you agree or whatever you’ve read that may be different is always informative. Hey, the more you read, the better off you’ll be. Just be careful to pick and choose the advice you follow wisely. Okay, anyway, we all know deca has a negative affect on your sex_drive. This also means that other esters of the drug such as the old school Laurabolin (nandrolone laurate) the eleven ester chain form of deca will have a similar affect if used in a similar dosage pattern. The half-life is very close to deca but exceeds it by a few days. The literature often says two weeks or more, but from experience, I’d say deca five to six days, and laurabolin, eight to ten. The shorter acting versions such an nandrolone phenylpropionate will also have the same affect. Also, the onset of phenylprop will be faster which will probably cause you to notice a decreased sex_drive earlier in your cycle. Remember that many side affects with anabolic steroids are duration dependent as well as dosage dependent and you may not always experience the effects listed in the information sources you read and study.
Trenbolone is interesting because much of what you read will end in two truths. Number one, it is androgenic (some literature actually says six times more androgenic thatn testosterone), and two, it is a progesterone derived drug. This sounds like a paradox. For functional purposes, tren is a hearty androgen that also has a chemical structure that is very similar to progesterone. Trenbolone is also non-aromatizing. The real world application is that tren can make you strong and grow like you are on test, without the water weight (there will be no estrogen), and without the increased sex_drive caused by testosterone (because of tren’s anti-libido progestagenic affect).
II. Aromatizing drugs affect your natural testosterone production by interfering with the classic feed-back loop. Most know that hight estrogen levels affect your pituitary gland and shuts down your body’s natural production of testosterone. The pituitary gland is a master regulator of hormonal levels. Actually, it is controlled by the true master gland, the hypothalamus…but this can get confusing and will take us off topic. For now, just know that the pituitary senses high estrogen or progesterone, and shuts off a hormone called leutinizing hormone or LH. LH travels from the pituitary to the testes, finds specific cells called Leydig cells, and tells the Leydig cells to kick out some test. If you interefere with this message, you interfere with natural testosterone production. This is called the hypo-thalamic-pituitary-testicular-axis. All this means is a loop that senses estrogen or progesterone levels for the purpose of regulating testosterone. This is also the very loop that makes anti-estrogenic drugs effective at increasing your testosterone levels post cycle.
Once your system is clean from anabolics (days to weeks depending on the dosage and drugs used), use of anti-estrogens work to block the estrogen from the pituitary, thereby telling the pituitary, “hey, we don’t have enough testosterone round these parts, fire some up there Billy Bob”. The pituitary then sends/ allows LH to be produced once again where it then travels to the Leydig cells of the testes, and stimulates the machinery to turn back on. You will know when this happens because this is when you can first tell that your balls are getting bigger again, your energy level comes back up, you start to feel good again since being off, etc. Common drugs that aromatize include the nandrolones, dianabol and all the testosterone esters. The rest of the anabolic steroids are constantly disagreed upon amongst gurus, and their outcomes are not definitive enough to mention in detail, whether you are considering the likelihood of chemical structures, or empirical real world effects. After all, deca, test and d-bol are the drugs most used that will be the major culprits with regards to aromatization. I know, you all have probably known someone who has gotten some nasty estrogenic type side effects from some other drug besides deca, test or d-bol. Those side effects come from other root causes…However you look at it, estrogenic affects from anabolic steroids serve to decrease sex drive by shutting off your natural supply.
Only if the drugs are androgenic enough and affect the sexual areas of your brain in just the right way will the drug cancel its own affects and increase sex_drive. Your individual biochemistry comes into play with many of the sexual highs and lows of anabolic steroid usage, it takes individual experience to know what you need and when. But the information found here will hopefully take much of the guesswork out of your experiences.
III. Progestagenic Affects of drugs are often over-looked. This is because most individuals do not know how potent progesterone really is in the body. All you ever really here is the bad stuff about estrogen. Well, progesterone my friends can be pretty nasty. Remember what we've said about sex offenders and progesterone. the govenment itself uses progesterone treatment to kill the sex_drive of male sex offenders in order to help control their cravings. If it works on a freak, it will definitely work on a normal person. Don't forget, progesterone kills sex_drive in the male.
As an important side note...I’ve tried hard to not believe everything I’ve ever read or studied unless there is:
1. Good and reasonable scientific rationale (not necessarily proof though, legitimate scientific tests always lag way behind empirical evidence)
2. Empirical Evidence
By good scientific rationale, I am referring to a logical reason(s) for a given occurrence to take place. Fore example, with progesterone, it is known that the male breast contains receptors for progesterone. This is known by testing of subjects who have clinical gynocomastia from causes other than anabolic steroids. Also, progesterone is meant to sensitize the body’s estrogen receptors to estrogen. Everyone knows that anadrol (a potent progestagenic anabolic steroid) can play a key role in the development of gyno. However we now know as fact that drol cannot aromatize due to its chemical structure. It is impervious to the enzyme aromatase which is the only enzyme that can do this job. And yet, drol can still cause gyno, right? Well, not exactly.
The two reasons why drol can influence gyno have already been said, but have not been grouped together. The reasons are:
1. Anadrol has a potent progestagenic affect.
2. Anadrol may sensitize estrogen receptors to estrogen.
With respect to drug potency and effectiveness you can either increase the amount of drug available, or increase the sensitivity of the receptor(s). In the case of anadrol, the drug both acts sufficiently to affect progesterone receptors, and acts to sensitize estrogen to its own estrogen receptors.
Trenbolone also has progestagenic affects. Tren, as we’ve previously said, possesses strong androgenic properties. Tren is not known in the anabolic steroid community to cause gyno. However, it would be interesting to observe many cycles of anabolic steroids which include a heavily aromatizing steroid with trenbolone. No doubt many athletes have combined tren and testosterone with miraculous results. It would be interesting to note amongst these individuals if they have ever developed gyno when combining the drugs, but not when taking them alone. Remember, progesterone sensitizes the body to estrogen, so whatever estrogen is available will be more likely to act out and cause the negative effects we know estrogen is responsible for.
The moral of the story is to be cautious and knowledgeable about the drugs you are combining. Actually, that should be true whether you are 21 and raving at a night club on e, or a responsible individual, through with your party stage, and maximizing your safe use of anabolic steroids. Drug synergy can be an amazingly useful tool when used for maximal gain, but can be very unfortunate and even dangerous if multiple negative properties of drugs are allowed to gather inside the body. We will talk more about the specifics of drug synergy in another section.